MiR-29 Induces K562 Cell Apoptosis by Down-Regulating FoxM1

نویسندگان

  • Xiaofang Wang
  • Hua Zhong
  • Lei Wang
  • Yuqian Dong
  • Ankui Jia
  • Qingjiang Mo
  • Chenguang Zhang
چکیده

BACKGROUND Leukemia seriously threatens human life and health. MicroRNAs can regulate cell growth, proliferation, and death. This article investigated the role of miR-29 on regulating leukemia cell growth, proliferation, and apoptosis. MATERIAL AND METHODS miR-29 and scramble miRNA were transfected to K562 cells. MTT assay, colony formation assay, caspase-3 activity detection, and flow cytometry were applied to test miR-29 effect on cell growth, proliferation, and apoptosis. Western blot was used to detect Forkhead box protein M1 (FoxM1) protein expression. After we transfected miR-29, K562 cells were transfected with FoxM1 siRNA to test cell apoptosis. RESULTS K562 cell growth and proliferation were inhibited after transfection with miR-29. Apoptosis phenome and caspase-3 activation were observed. FoxM1 level decreased. SiRNA FoxM1 enhanced miR-29-induced K562 cell apoptosis. FoxM1 overexpression suppressed miR-26-induced K562 cell apoptosis. CONCLUSIONS MiR-29 restrained K562 cell growth and proliferation. MiR-29 induced K562 cell apoptosis through down-regulating FoxM1.

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عنوان ژورنال:

دوره 21  شماره 

صفحات  -

تاریخ انتشار 2015